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Anabolic steroids are a class of natural and synthetic steroid hormones that promote cell growth and division, resulting in growth of several types of tissues, especially muscle and bone. Acetone is the main active substance. In a variety of studies, many studies have been conducted regarding the effects of muscle hypertrophy and, particularly, the effects of synthetic steroids on this growth. These studies were reported to show a significant increase in muscle mass, and possibly also increases in muscle cell mass within certain muscle cells. Such findings may contribute to increased muscle strength and other functional adaptations during and after periods of training, particularly during lean phase of a muscle training program. In the present study, the effect of acute acute treatment with 2 mg/kg of acetic acid was evaluated in female rats treated with vehicle or testosterone. Acetyl-CoA carboxylase (ACC) activity was determined in muscle, hair follicle, bone, striatum and adipose tissue. Acetyl-CoA carboxylase has been shown to increase in the serum of male rodents treated with high doses of testosterone and to be correlated with muscle mass (e.g. Lipsinger et al., 1988a). Studies involving acute acute acute treatment with acetic acid had revealed an increase of 0.8% of total ACC activity (Cao and Fagard, 1987). Acetyl-CoA esterase activity was determined in brain at a concentration of about 2 mg/kg. Acetyl-CoA esterase is a catabolic enzyme which has been shown to increase in blood levels (DeVore and DeVore, 1991). These findings in brain tissue also had been reported in the literature when acetic acid was administered to normal rats or to mice, both with and without concurrent treatment with concurrent muscle growth and exercise (Lipsinger et al., 1988b). Studies have suggested that this increase in ACC activity in tissue tissue is not related with activation of this ACC enzyme either directly (e.g. by increased lipolytic activity), or indirectly via its inhibition (e.g. by lowered lipolytic activity) (deVore et al., 1992; Lipsinger et al., 1989). It is also noteworthy that in rats treated with acetic acid, the levels of lipid peroxidation increased as an effect of the drug, and the levels of fatty acid synthesis in these tissues also showed increases (Lipsinger et al., 1993). Since some investigators suggest that ACC is one target of testosterone treatment (see below) as a consequence of increased ACC activity (Chang et al., 1995), it will be of interest and relevance to determine whether any of the above discussed effects Related Article: